72300X Testfagen & 72300X Schulungsangebot - 72300X Tests - Veilleuse-Bebe

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NEW QUESTION: 1
Click the Exhibit button.

userehost# run show route
inet.0: 4 destinations, 4 routes (4 active, 0 holddown, 0 hidden)
+ = Active Route, - = Last Active, * = Both
0.0.0.0/0 *[Static/5] 00:05:06
> to 172.16.1.1 via ge-0/0/1.0
172.16.1.0/24 *[Direct/O] 00:05:06
> via ge-0/0/1.0
172.16.1.3/32 *[Local/0] 00:05:07
Local via ge-0/0/1.0
192.168.200.2/32 *[Local/0] 00:05:07
Reject
vr-a.inet.0: 2 destinations, 2 routes (2 active, 0 holddown, 0 hidden) + = Active Route, - = Last Active, * = Both
192.168.1.0/24 *[Direct/0] 00:01:05
> via ge-0/0/2.0
192.168.1.1/32 *[Local/0] 00:01:05
Local via ge-0/0/2.0
vr-b.inet.0: 2 destinations, 2 routes (2 active, 0 holddcwn, 0 hidden) + = Active Route, - = Last Active, * = Both
192.168.1.0/24 *[Direct/O] 00:01:05
> via go-0/0/3.0
192.168.1.1/32 *[Local/0] 00:01:05
Local via ge-0/0/3.0
User 1 will access Server 1 using IP address 10.2.1.1. You need to ensure that return traffic is able to reach User 1 from Server 1.
Referring to the exhibit, which two configurations allow this communication (Choose two.)
A. [edit security nat static]
[email protected]# show
rule-set server-nat {
from zone untrust;
rule 1 {
match {
destination-address 10.2.1.1/32;
}
then {
static-nat {
prefix {
192.168.1.2/32;
routing-instance vr-a;
}
}
}
}
}
B. [edit security nat static]
[email protected]# show
rule-set server-nat {
from zone [ untrust ];
rule 1 {
match {
destination-address 10.2.1.1/32;
}
then {
static-nat {
prefix {
192.168.1.2/32;
}
}
}
}
}
C. [edit security nat static]
[email protected]# show
rule-set in {
from zone untrust;
to zone cust-a;
rule overload {
match {
source-address 0.0.0.0/0;
}
then {
source-nat {
interface;
}
}
}
}
D. [edit security nat static]
[email protected]# show
rule-set server-nat {
from zone [ junos-host untrust ];
rule 1 {
match {
destination-address 10.2.1.1/32;
}
then {
static-nat {
prefix {
192.168.1.2/32;
routing-instance vr-b;
}
}
}
}
}
Answer: B,D

NEW QUESTION: 2
The qualities expected of a professional musician seem _______, for she must be studious, disciplined, and technically impeccable while bringing passion and _______ to each performance.
A. unattainable . . emotion
B. paradoxical . . spontaneity
C. multifarious . . virtuosity
D. eclectic . . impulsiveness
E. ambiguous . . capriciousness
Answer: B
Explanation:
Explanation/Reference:
Explanation:
Discipline, studiousness, and technical precision are generally thought of as contrary to "passion." So, the sentence suggests that a professional musician must possess contradictory or opposing qualities. The word paradoxical captures the sense of contradiction, while the spontaneity fits nicely with "passion" and contrasts appropriately with "disciplined."

NEW QUESTION: 3
Examine the following PL/SQL block:
SET SERVEROUTPUT ON
SET LONG 10000
ECLARE report clob;
BEGIN
report := DBMS_SPM.EVOLVE_SQL_PLAN_BASELINE();
DBMS_OUTPUT .PUT_LINE(report);
END;
Which statement describes the effect of the execution of the above PL/SQL block?
A. The plan baselines are verified with the SQL profiles.
B. The nonaccepted plans in the SQL Management Base are verified with the existing plan baselines.
C. All the nonaccepted SQL profiles are accepted into the plan baseline.
D. All fixed plan baselines are converted into nonfixed plan baselines.
Answer: B
Explanation:
Explanation/Reference:
Explanation:

NEW QUESTION: 4
Your patient is a 58-year-old male who presents with onset of severe substernal chest pain and shortness of breath. An ECG reveals an acute STEMI, and he is on his way to the cardiac catheterization suite for percutaneous coronary intervention.
Which of the following drugs used in acute coronary syndromes treated with PCI must undergo oxidation by hepatic P450 enzymes to an active form?
A. Ticlopidine
B. Warfarin
C. Eptifibatide
D. Clopidogrel
E. Aspirin
Answer: D
Explanation:
Explanation
Clopidogrel and ticlopidine are ADP receptor pathway inhibitors. The irreversible inhibition of the ADP- dependent pathway of platelet activation is thought to be the result of covalent modification and inactivation of the platelet P2Y ADP receptor. This receptor is coupled to the inhibition of adenylyl cyclase. Both drugs are prodrugs and undergo conversion to active metabolites in the liver. However, clopidogrel must undergo oxidation by hepatic P450 enzymes to its active form. This is significant because many drugs are metabolized the hepatic P450 enzymes, including statins, and clopidogrel may interact with these medications. Clopidogrel is a second-generation thienopyridine and ticlopidine is a first-generation thienopyridine. Both drugs are indicated in combination with aspirin to prevent stent thrombosis. Eptifibatide is a GPIIb-IIIa receptor antagonist that is used to treat unstable angina and non-ST segment elevation myocardial infarction.
Eptifibatide is also used to reduce ischemic events in patients who are undergoing percutaneous coronary intervention. The drug is a synthetic peptide that directly antagonizes the GPIIb-IIIa receptor on the platelet.
Aspirin is an antiplatelet drug that works by inhibition of synthesis of prostaglandins. Prostaglandin G2 is the result of a synthesis pathway that is activated by platelets and endothelial cells, and results in localized vasoconstriction and induction of platelet aggregation, as well as causing release of platelet granules. Warfarin is an anticoagulant that acts on vitamin K-dependent reactions in the coagulation pathway. Vitamin K is necessary for hepatic synthesis of coagulation factors II, VII, IX and X, protein C and protein S.
Vitamin K- dependent carboxylation is necessary for induction of enzymatic activity of these coagulation factors. Take- home message: Clopidogrel, a second-generation thienopyridine ADP receptor pathway inhibitor, is indicated in combination with aspirin to prevent stent thrombosis in patients who undergo percutaneous coronary intervention after myocardial infarction. Clopidogrel is a prodrug that must undergo oxidation by hepatic P450 enzymes, and therefore may affect the activity of statins and other drugs dependent on the hepatic P450 enzymes.


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